Cardiomyopathy Ephakamileyo

Kulokhuqina kwe-cardiomyopathy, udonga lwenhliziyo oluyisigxobo oluthiwa yi-septum luhlala lubuhlungu kunokujwayelekile. Kodwa ukukhuluphala kungenzeka kunoma iyiphi indawo ekamelweni eliphansi lasehlangothini lobunxele lenhliziyo, okuthiwa yi-left ventricle.

I-Hypertrophic cardiomyopathy (HCM) yisifo lapho isigxobo senhliziyo siqina khona, okuthiwa yi-hypertrophied. Isigxobo senhliziyo esiqinile singenza kube nzima enhliziyweni ukupompa igazi.

Abantu abaningi abane-hypertrophic cardiomyopathy abazi ukuthi bayitholile. Lokho kungenxa yokuthi banezimpawu ezimbalwa, uma kukhona. Kodwa kubantu abambalwa abane-HCM, isigxobo senhliziyo esiqinile singabangela izimpawu ezinkulu. Lezi zihlanganisa ukuphefumula kanzima nobuhlungu esifubeni. Abanye abantu abane-HCM banokuguquka ohlelweni lwe-elekthrikhi lenhliziyo. Lezi zinguquko zingabangela ukushaya kwenhliziyo okungajwayelekile okusongela impilo noma ukufa okungazelelwe.

Izimpawu ze-hypertrophic cardiomyopathy zingafaka okukodwa noma ngaphezulu kwalokhu okulandelayo: Ubuhlungu obusengifubeni, ikakhulukazi ngesikhathi sokuzivocavoca. Ukuhlupheka, ikakhulukazi ngesikhathi noma ngemuva nje kokuzivocavoca noma eminye imisebenzi yomzimba. Ukuzwa okusheshayo, okuphephezela noma okushayayo kwezinhliziyo okuthiwa yi-palpitations. Ukuqina komoya, ikakhulukazi ngesikhathi sokuzivocavoca. Izimo eziningi zingabangela ukuqina komoya kanye nokushaya okusheshayo, okushayayo kwezinhliziyo. Kubalulekile ukuhlolwa ngokushesha ukuthola imbangela nokubhekana nokunakekelwa okufanele. Bheka ochwepheshe bakho bezempilo uma unemlando womndeni we-HCM noma noma iyiphi impango ehambisana ne-hypertrophic cardiomyopathy. Shaya u-911 noma inombolo yakho yokuphuthuma kwendawo uma unezinye zezimpawu ezilandelayo isikhathi esingaphezu kwemizuzu embalwa: Ukushaya kwenhliziyo okusheshayo noma okungajwayelekile. Ukuphefumula okuthanda ukukhathazeka. Ubuhlungu obusengifubeni.

Izimo eziningi zingabangela ukuphefumula kanzima kanye nokushaya kwenhliziyo okusheshayo, okunamandla. Kubalulekile ukuhlolwa ngokushesha ukuthola imbangela nokuthola ukunakekelwa okufanele. Bheka ochwepheshe bakho bezempilo uma unemlando womndeni we-HCM noma noma yiziphi izimpawu ezihlobene ne-hypertrophic cardiomyopathy.

Shaya u-911 noma inombolo yakho yephuthumayo yendawo uma unezinye zezimpawu ezilandelayo isikhathi esingaphezu kwemizuzu embalwa:

• Ukushaya kwenhliziyo okusheshayo noma okungajwayelekile.
• Ukuphefumula okunzima.
• Ubuhlungu esifubeni.

I-hypertrophic cardiomyopathy, ngokuvamile ibangelwa izinguquko ezinjini ezingenza ukuba ihlathi lenhliziyo liqine.

I-hypertrophic cardiomyopathy ngokuvamile ithinta udonga oluphakathi kwamakamelo amabili angaphansi enhliziyo. Lodonga lubizwa ngokuthi i-septum. Lamakamelo abizwa ngokuthi ama-ventricles. Udonga oluqine lungase luvimbele ukugeleza kwegazi liphume enhliziyweni. Lokhu kubizwa ngokuthi i-obstructive hypertrophic cardiomyopathy.

Uma kungekho ukuvimbela okukhulu kokugeleza kwegazi, isimo sibizwa ngokuthi i-nonobstructive hypertrophic cardiomyopathy. Kodwa ikhamera ephambili yokupompa yenhliziyo, ebizwa ngokuthi i-left ventricle, ingase iqine. Lokhu kwenza kube nzima enhliziyweni ukuphumula. Ukuqina kwenza futhi kunciphise inani legazi elihlala e-ventricle futhi lithunyelwe emzimbeni ngesikhathi ngasinye sokushaya kwenhliziyo.

Amaseli ehlathi lenhliziyo futhi ahlelwa ngendlela ehlukile kubantu abane-hypertrophic cardiomyopathy. Lokhu kubizwa ngokuthi i-myofiber disarray. Kungase kubangele ukushaya kwenhliziyo okungajwayelekile kwabanye abantu.

I-hypertrophic cardiomyopathy ivame ikluliwe emindenini. Lokho kusho ukuthi iyefa. Abantu abanomzali oyedwa one-hypertrophic cardiomyopathy banesilinganiso sama-50% sokuthola ushintsho lwe-jini oluyimbangela yesifo.

Abazali, abantwana, noma abafowabo nodadewabo bomuntu onayo i-hypertrophic cardiomyopathy kufanele babuze iqembu labo lokunakekelwa kwezempilo ngokuhlolwa kokuxilongwa kwesifo.

Izinkinga ze-hypertrophic cardiomyopathy zingabandakanya:

• Ukubamba okungajwayelekile kwenhliziyo (AFib). Imisipha yenhliziyo ebukhulu kakhulu kanye nezinguquko esakhiweni sezakhi zehliziyo zingabangela ukushaya kwenhliziyo okungajwayelekile futhi ngokuvamile okusheshayo kakhulu okuthiwa yi-AFib. I-AFib iphinde ikhulise ingozi yezigaxa zegazi, ezingahamba ziye ebuchosheni futhi zibangele isiguli.
• Ukugadwa kwegazi. Kubantu abaningi, imisipha yenhliziyo ebukhulu kakhulu ivala ukugadwa kwegazi okuphuma enhliziyweni. Lokhu kungabangela ukuphefumula kanzima ngenkathi usebenza, ubuhlungu ebeleni, ukukhungatheka nokuhlanza.
• Isifo se-Mitral valve. Uma imisipha yenhliziyo ebukhulu kakhulu ivala ukugadwa kwegazi okuphuma enhliziyweni, i-valve phakathi kwamakamelo enhliziyo yangakwesobunxele ingase ingavalwa kahle. Leyo valve ibizwa nge-mitral valve. Uma ingavalwa kahle, igazi lingase liphume emuva ekamelweni eliphezulu elingakwesobunxele. Lokhu kuyisimo esibizwa ngokuthi yi-mitral valve regurgitation. Kungenza izimpawu ze-hypertrophic cardiomyopathy zibe zimbi kakhulu.
• I-Dilated cardiomyopathy. Kubenani elincane labantu abane-HCM, imisipha yenhliziyo ebukhulu kakhulu iba buthakathaka futhi ayisebenzi kahle. Isimo siqala ekamelweni elingaphansi elingakwesobunxele lenhliziyo. Ikamelo liba likhulu. Inhliziyo iphompa ngamandla ambalwa.
• Ukwehluleka kwenhliziyo. Ngokuhamba kwesikhathi, imisipha yenhliziyo ebukhulu kakhulu ingaba lukhuni kakhulu ukugcwalisa inhliziyo ngegazi. Ngenxa yalokho, inhliziyo ayikwazi ukuphompa igazi elanele ukuhlangabezana nezidingo zomzimba.
• Ukuhlanza, okuthiwa yi-syncope. Ukushaya kwenhliziyo okungajwayelekile noma ukugadwa kwegazi okuvinjiwe ngezinye izikhathi kungabangela ukuhlanza. Ukuhlanza okungachazwanga kungahlobana nokufa kwesikhathi esifushane senhliziyo, ikakhulu uma kwenzeke muva nje futhi kumuntu osemncane.
• Ukufa kwesikhathi esifushane senhliziyo. Ngokungavamile, i-hypertrophic cardiomyopathy ingabangela ukufa kwesikhathi esifushane okuxhumene nenhliziyo kubantu bazo zonke izinsuku. Abantu abaningi abane-hypertrophic cardiomyopathy abazi ukuthi bayitholile. Ngenxa yalokho, ukufa kwesikhathi esifushane kwenhliziyo kungaba uphawu lokuqala lesimo. Kungenzeka kubantu abasebasha ababonakala benempilo, kufaka phakathi abadlali bezemidlalo esikoleni esiphakeme nabanye abantu abasebasha, abasebenza kanzima.

Asikho isindlela eyaziwayo yokuvimbela i-hypertrophic cardiomyopathy (HCM). Kubalulekile ukuthola isimo ngokuhlolwa ngokushesha okukhulu ngangokunokwenzeka ukuqondisa ukwelashwa nokuvimba izinkinga. I-hypertrophic cardiomyopathy ngokuvamile idluliselwa emindenini. Uma unawo umzali, umfowethu, udade noma ingane enenkinga ye-hypertrophic cardiomyopathy, buza iqembu lakho lokunakekelwa kwezempilo uma ukuhlolwa kwe-genetic kulungele wena. Kodwa-ke akusibo bonke abantu abane-HCM abanokuguquka kwe-gene okutholakala ngokuhlolwa. Futhi, amanye ama-insurance companies angase angawagcini ukuhlolwa kwe-genetic. Uma ukuhlolwa kwe-genetic kungenziwa, noma uma imiphumela ingawusizi, ukuhlolwa kungase kwenziwe nge-echocardiograms eziphindaphindwayo. I-echocardiograms isebenzisa amaza omsindo ukwenza izithombe zenhliziyo. Kubantu abanolunga lomndeni olune-hypertrophic cardiomyopathy: - Ukuhlolwa kwe-echocardiogram kunconywa kusukela eminyakeni ecishe ibe yi-12. - Ukuhlolwa nge-echocardiograms kufanele kuqhubeke unyaka ngamunye kuya kweminyaka emi-3 phakathi kweminyaka engu-18 kuya kwengu-21. - Ngemva kwalokho, ukuhlolwa kungase kwenziwe eminyakeni emihlanu yonke phakathi kokukhula. Ungase udinga ukwenza i-echocardiogram kaningi ngokuya ngempilo yakho jikelele nokukhetha kweqembu lakho lokunakekelwa kwezempilo.

Uchwepheshe wezempilo ukhomba bese ulalela inhliziyo yakho nge divayisi ebizwa ngokuthi yi-stethoscope. Kungase kuzwakale umsindo ongaphambene lapho ulalela inhliziyo.

Ulungu lweqembu lakho lezeMpilo ngokuvamile libuza imibuzo mayelana nezimpawu zakho kanye nomlando wakho wezokwelapha nowomndeni. Ukuhlolwa kwe-genetic noma ukwelulekwa kungase kunconywe uma unomlando womndeni wesimo.

Kwenziwa ukuhlolwa ukuhlola inhliziyo nokufuna izimbangela zanoma yiziphi izimpawu.

• Echocardiogram. I-echocardiogram ivame ukusetshenziswa ukuxilonga i-hypertrophic cardiomyopathy. Amaza omsindo asebenza ukwenza izithombe zenhliziyo ebethayo. Lo mkhuba ubonisa ukuthi amakamelo nenhliziyo isebenza kangcono kangakanani. I-echocardiogram ingabona futhi uma i-muscle yenhliziyo ibukhulu kunalokho okufanele kube.
• Electrocardiogram (ECG noma EKG). Lo mkhuba osheshayo futhi ongenaphimbo uqapha umsebenzi wamagetsi wenhliziyo. Izicucu ezinamathelayo ebizwa ngokuthi yi-electrodes zibekwa esifubeni kanye ngezinye izikhathi ezandleni nasezinzwaneni. Izingcingo zixhuma i-electrodes kukhompyutha, ephrinta noma ebonisa imiphumela yokuhlolwa. I-ECG ingabonisa ukushaya kwenhliziyo okungajwayelekile kanye nezibonakaliso zokukhuluphala kwenhliziyo.
• Holter monitor. Le divayisi encane, ephathekayo ye-ECG irekhoda umsebenzi wenhliziyo. Igqokwe usuku noma amabili ngenkathi wenza imisebenzi yakho evamile.
• Cardiac MRI. Lo mkhuba usebenzisa ama-magnets anamandla namaza omsakazo ukwenza izithombe zenhliziyo. Unikeza ulwazi mayelana ne-muscle yenhliziyo nokuthi inhliziyo nevalvu zenhliziyo zisebenza kanjani. Lo mkhuba uvame ukwenziwa nge-echocardiogram.
• Ukuhlolwa kokucindezela. Ukuhlolwa kokucindezela ngokuvamile kuhilela ukuhamba etreadmill noma ukuqhubela ibhayisikili elimi ngenkathi inhliziyo iqapha. Ukuhlolwa kokucindezela komzimba kusiza ukwembula ukuthi inhliziyo iphendula kanjani emisebenzini yomzimba.
• Cardiac CT scan. Ngokungavamile, lo mkhuba wenziwa ukuxilonga i-hypertrophic cardiomyopathy. Kodwa kungase kunconywe uma i-MRI ingasetshenziswa. I-Cardiac CT scan isebenzisa i-X-rays ukwenza izithombe zenhliziyo nesifuba. Ingabonisa usayizi wenhliziyo.

Imigomo yokwelapha i-cardiomyopathy ephakamileyo iwukunciphisa izimpawu nokuvimba ukufa kwenhliziyo okuzumayo kubantu abasezingeni elikhulu lobungozi. Ukwelashwa kuncike ekuthini ukukhulu kangakanani izimpawu. Uma une-cardiomyopathy futhi ukhulelwe noma ucabanga ngokukhulelwa, khuluma nodokotela wakho. Ungase uqondiswe kudokotela onolwazi ekukhulelweni okungozini enkulu. Lo dokotela angaba yi-perinatologist noma uchwepheshe wezokwelapha umama-nesisu. Imithi Imithi ingasiza ukunciphisa indlela umsipha wenhliziyo ocindezela ngayo nokuthambisa izinga lenhliziyo. Ngaleyo ndlela, inhliziyo ingapompa igazi kangcono. Imithi yokwelapha i-cardiomyopathy ephakamileyo kanye nezimpawu zayo ingafaka: Abaqaphi be-beta njenge-metoprolol (Lopressor, Toprol-XL), i-propranolol (Inderal LA, Innopran XL) noma i-atenolol (Tenormin). Abaqaphi be-calcium channel njenge-verapamil (Verelan) noma i-diltiazem (Cardizem, Tiazac, nabanye). Imithi ebizwa nge-mavacamten (Camzyos) eunciphisa ukucindezeleka enhliziyweni. Ingalapha i-HCM ephakamileyo kubantu abadala abanezibonakaliso. Iqembu lakho lokwelapha lingase liqondise le mithi uma ungakwazi ukuthatha noma awulunge nge-beta blockers noma i-verapamil. Imithi yengcindezi yenhliziyo njenge-amiodarone (Pacerone) noma i-disopyramide (Norpace). Abaphuthuma igazi njenge-warfarin (Jantoven), i-dabigatran (Pradaxa), i-rivaroxaban (Xarelto) noma i-apixaban (Eliquis). Abaphuthuma igazi bangasiza ukuvimba ama-blood clots uma une-atrial fibrillation noma uhlobo lwe-apical le-hypertrophic cardiomyopathy. I-Apical HCM ingandisa ingozi yokufa kwenhliziyo okuzumayo. Ukuhlinzwa noma ezinye izinqubo I-Septal myectomy Khulisa isithombe Vala I-Septal myectomy I-Septal myectomy I-Septal myectomy ukwelashwa kwenhliziyo evulekile. Udokotela ukhumula ingxenye yesipha esikhulu, esikhulu phakathi kwamakamelo aphansi enhliziyo abizwa ngama-ventricles, njengoba kuboniswe enhliziyweni ngakwesokudla. I-Apical myectomy Khulisa isithombe Vala I-Apical myectomy I-Apical myectomy I-Apical myectomy ukwelashwa kwenhliziyo evulekile lokwelapha i-cardiomyopathy ephakamileyo. Udokotela ukhumula umsipha wenhliziyo oqine kakhulu eduze nokuma komphetho wenhliziyo. Kunezindlela eziningi zokuhlinzwa noma izinqubo ezitholakalayo zokwelapha i-cardiomyopathy noma izimpawu zayo. Zifaka: I-Septal myectomy. Lokhu kusebenza kwenhliziyo evulekile kungase kunconywe uma imithi ingalungisi izimpawu. Kuhilela ukususa ingxenye yodonga oluqine kakhulu, olukhulu phakathi kwamakamelo enhliziyo. Lodonga lubizwa ngokuthi i-septum. I-Septal myectomy iyasiza ekuthuthukiseni ukusabalala kwegazi ngaphandle kwenhliziyo. Iphinde inciphise ukusabalala emuva kwegazi nge-mitral valve. Ukuhlinzwa kungenziwa ngezindlela ezahlukene, kuye ngendawo yomsipha wenhliziyo oqine kakhulu. Kulolu hlobo, oluthiwa yi-apical myectomy, ochwepheshe bokuhlinzwa bakhumula umsipha wenhliziyo oqine kakhulu eduze nokuma komphetho wenhliziyo. Ngezinye izikhathi i-mitral valve ilungiswa ngesikhathi esifanayo. I-Septal ablation. Le nqubo isebenzisa utshwala ukunciphisa umsipha wenhliziyo oqine kakhulu. Umphako omde, onobunono obubizwa ngokuthi i-catheter ubekwa emithanjeni ehlinzeka igazi endaweni ethintekayo. Uthwala ludlula ngemphako. Ukushintsha ohlelweni lokukhombisa amandla kwenhliziyo, okuthiwa i-heart block, kuyinkimbinkimbi. I-heart block kufanele iwelashwe nge-pacemaker. Idivayisi encane ibekwa esifubeni ukuze isize ukulawula ukushaya kwenhliziyo. I-Implantable cardioverter-defibrillator (ICD). Le divayisi ibekwa ngaphansi kwesikhumba eduze kwentamo. Ihlola njalo ingcindezi yenhliziyo. Uma idivayisi ithola ukushaya kwenhliziyo okungajwayelekile, ithumela imithambeka yamandla aphansi noma aphezulu ukuze ibuyekeze ingcindezi yenhliziyo. Ukusetshenziswa kwe-ICD kuye kwabonakala kusiza ukuvimba ukufa kwenhliziyo okuzumayo, okuvela kubantu abambalwa abane-hypertrophic cardiomyopathy. I-Cardiac resynchronization therapy (CRT) device. Ngokuvamile, le divayisi efakwe isetshenziswa njengokwelapha i-hypertrophic cardiomyopathy. Ingasiza amakamelo enhliziyo ukucindezela ngendlela ehlelile futhi isebenza kahle. I-Ventricular assist device (VAD). Le divayisi efakwe isetshenziswa kancane ukwelapha i-hypertrophic cardiomyopathy. Iyasiza ukusabalala kwegazi ngenhliziyo. Ukuhlinzwa kwenhliziyo. Lokhu ukwelashwa kwenhliziyo okuhlinzwa ukuze kuhlanganiswe inhliziyo egula ngenhliziyo enempilo yomnikelo. Kungaba yindlela yokwelapha ukwehluleka kwenhliziyo ekuqedeni uma imithi nezinye izindlela zokwelapha zingasebenzi. I-Hypertrophic cardiomyopathy nezinketho zokwelapha Dlala Dlala Buyela kuvidiyo 00:00 Dlala Sesha amasekhondi angu-10 emuva Sesha amasekhondi angu-10 phambili 00:00 / 00:00 Thulisa Izilungiselelo Isithombe esisesithombeni Isikrini esigcwele Bonisa umbhalo wevidiyo I-Hypertrophic cardiomyopathy nezinketho zokwelapha U-Steve R. Ommen, M.D., Izifo zeCardiovascular, i-Mayo Clinic: I-hypertrophic cardiomyopathy isimo esingakaze siqondwe kahle futhi esesaba kakhulu emhlabeni jikelele. E-United States kuphela, kunabantu abangaphezu kwesigamu sezigidi abane-hypertrophic cardiomyopathy, abaningi babo abanawo nhlobo izimpawu futhi abazi ngokuxilongwa kwabo. Abanye abantu bangase bafe ngokuzumayo. Ukufa kwenhliziyo okuzumayo kwenzeka ngokungahleliwe ngaphandle kokuxwayisa. U-Hartzell V. Schaff, M.D., Ukuhlinzwa kwenhliziyo, i-Mayo Clinic: Ngaphezu kwe-2/3 yabagula bayoba nokuvimba. Futhi ukuvimba endleleni yokusabalala kwegazi kwesobunxele kuyisibonakaliso sokuhlinzwa kubantu abanezibonakaliso. Ngakho-ke manje siyazi ukuthi i-2/3 yabagula abane-hypertrophic cardiomyopathy nokuvimba bangabakhethi bokuhlinzwa. UDkt. Ommen: I-Hypertrophic cardiomyopathy iwumkhuhlane ovamile owudluliselwa ngezizukulwane we-cardiomyopathy noma isifo somsipha wenhliziyo. Abantu bazalwa benayo, kodwa i-hypertrophy ayibonakali iqala ukuthuthuka kuze kube ebusheni, ukukhula okukhulu, noma ngaphezulu. Kungenzeka ukuthi izingane zizalwe zinemisipha yenhliziyo ehlwempu, kodwa lokho kuyinto engavamile futhi ngokuvamile izimpawu ezinzima zesifo. Futhi kuye kwachazwa njengokungabi khona kuze kube yilapho abantu beseminyakeni yabo emihlanu noma eyisithupha yokuphila. Ngakho-ke, impela, ukuqala kungaba kunoma yisiphi isikhathi sokuphila. Futhi ngokuqinisekile izimpawu zingase zivele ekuphileni konke. UDkt. Schaff: Izimpawu ezivamile abantu abazithola uma bene-obstructive hypertrophic cardiomyopathy ukuphefumula buhlungu, ubuhlungu besifuba obunjenge-angina nokulahlekelwa umqondo. Ngokudabukisayo, ezinye zalezi zimpawu zithuthuka kancane futhi isikhathi eside kangangokuthi abantu abaziqondi kahle ukuthi banqunywe kangakanani. UDkt. Ommen: Kubantu abanezibonakaliso ngenxa ye-hypertrophic cardiomyopathy, umugqa wokuqala wokwelapha ukusebenzisa ukuphathwa kwezokwelapha, imithi. Ngokuvamile, lokho ukwengeza imithi ethile, kodwa ngezinye izikhathi abantu basebenzisa imithi engabenza babe sibi kakhulu. Ngakho-ke ezinye zezindlela zokwelapha eziphumelelayo ukususa izinto ezingalungile, bese mhlawumbe kufanele kwengezwe izinto ezifanele ukuze zibasize ngezimpawu zabo ngokuhamba kwesikhathi. Kubantu abangaphenduli kulezo zinguquko zezokwelapha, noma kulabo abathintekile imithi ebangela imiphumela emibi engakhuthali, khona-ke yilapho siqhubekela ezintweni ezifana nokuhlinzwa kwe-myectomy, okuthuthukisa izimpawu zabo ngokuqinile. UDkt. Schaff: Abantu abaqondiswa ekuhlinzweni cishe bahlulekile ukwelashwa kwezokwelapha noma banezinto ezingathandeki ezivela emithini ebanqunyelela njengoba nje izimpawu ze-hypertrophic cardiomyopathy. Ngakho-ke ukuhlinzwa ukuze kuqediwe ukuvimba indlela yokusabalala kwegazi ukuze kuqediwe izimpawu. Futhi kwabanye abantu, ukuze bavunyelwe ukuyeka imithi enezinto ezingathandeki. UDkt. Ommen: Ukuhlinzwa kwe-myectomy kuye kwaba ukwelashwa okuphumelelayo kakhulu kwabantu abaningi. Noma kunjalo, akusetshenziswa kakhulu njengoba kungenzeka ngenxa yezimvo zangaphambili mayelana nengozi ephakeme ngokwelashwa, ukungatholakali kwabachwepheshe bokuhlinzwa abangakwenza. Kepha ezandleni zezindawo eziqeqeshwe kahle, amanani okuhlupheka aphansi kakhulu futhi amanani ethu okuphumelela aphezulu kakhulu. UDkt. Schaff: Manje senza i-septal myectomy ebanzi kakhulu esondela ekupheleni kwenhliziyo. Futhi sifunde eminyakeni edlule ukuthi yingxenye yale myectomy ekude kakhulu ebaluleke kakhulu ngokuqeda izimpawu. Abantu abambalwa abaye baba nokuhlinzwa kwesibili, abaqondiswe kithi ngemuva kokuba nokuhlinzwa okungalunganga, sifumene ukuthi i-myectomy ayizange iqhubekeke kakhulu esiswini. Akuyona impela ukukhula kabusha kwemisipha. Kuyinto nje yokwelapha okungalunganga ekuqaleni. UDkt. Ommen: Ngokuhlinzwa kwe-myectomy, udokotela ukhumula ingxenye yesipha esikhulu se-septum, esiqinisa indlela yokuhamba kwegazi, ngaphandle kwenhliziyo. Ngokwenza lokhu, kushintsha indlela igazi elihamba ngayo esiswini. Kuvumela i-mitral valve ukusebenza ngokujwayelekile. Futhi kuvumela igazi ukushiya inhliziyo ngaphandle kokwandisa ukucindezela noma ukwandisa amandla. Lo msipha awukhuliswa kabusha ngokuhamba kwesikhathi. Uyilungiso eliphelele. UDkt. Schaff: Sifumene ukuthi kuncane kakhulu ukwenza okuthile kwi-mitral valve. Futhi ingozi yokwenza okuthile kwi-mitral valve, lapho kuvela ukuthi akudingeki, ukuthi kunezithuba zokulimala. Ngakho-ke singathanda ukwenza i-septal myectomy, siphume ekuhlinzweni, sihlole i-mitral valve nge-echocardiogram ngesikhathi sokuhlinzwa, ngaphambi kokubhekana ne-mitral valve uma kunokubuyela emuva. Singabona uma ukubuyela emuva kwe-mitral kuqediwe ngokushesha ngemuva kwe-myectomy uma i-aorta ivaliwe futhi inhliziyo iqale kabusha. I-echocardiogram yenziwa ekamelweni lokuhlinzwa futhi siyazi ngokushesha uma ukubuyela emuva kwe-mitral kuqediwe. Ukuhlinzwa kutholakala kwabanye abantu abane-non-obstructive hypertrophic cardiomyopathy. Futhi laba bangabantu abanohlu lwe-hypertrophy. Abanye balabo bantu bane-diastolic heart failure ehambisana namakamelo amancane kakhulu e-ventricular. Futhi kulaba bantu, ukwenza i-transapical myectomy ukukhulisa i-ventricle kungathuthukisa izimpawu zabo zokwehluleka kwenhliziyo. UDkt. Ommen: Ngenkathi sibona imiphumela emihle evela ekuhlinzweni kwe-myectomy njengoba kwenziwa manje, kusengento okufanele yenziwe kuphela ezindaweni eziqeqeshwe kahle. Imininingwane yakamuva ephume isikisela ukuthi ezindaweni ezincane, eziphakathi, ngisho nezingathiwa “ezinkulu,” kunobunzima bokufa, okusho ukuthi luphezulu kakhulu ezindaweni ezincane futhi oluphansi kakhulu ezindaweni ezinkulu. Kepha ngisho nalabo abathiwa izindawo ezinkulu banamanani okufa aphezulu kakhulu kunalokho okubikwa ezindaweni eziqeqeshwe kahle. Futhi lokhu kuyinqubo okufanele yenziwe yilabo abajwayelene kakhulu nale nqubo, futhi benza okuningi kwabo. UDkt. Schaff: E-Mayo Clinic, senza ukuhlinzwa okungaphezu kuka-3,000 kwe-hypertrophic cardiomyopathy. Senza ukuhlinzwa okungu-200 kuya ku-250 unyaka ngamunye. Ukufa ngenqubo kungaphansi kuka-1%, ikakhulu kubantu abaphilile. UDkt. Ommen: Enye yezingxenye ezinkulu yayo yonke inkulumo engiyenza nabantu ukubasiza baqonde ukuthi yini ingozi yabo yokufa kwenhliziyo okuzumayo, nokuthi bangase bacabange ngokuba ne-implantable defibrillator. Abantu bethu abaye bahlinzwa banamanani aphansi okufa kwenhliziyo okuzumayo namanani aphansi we-defibrillators yabo ukuqhuma kulabo abaye baba nayo. UDkt. Schaff: Enye yezinto esizifundile ngemuva kokwenza i-septal myectomy ukuthi impela ukwanda kwe-ventricular arrhythmia kubonakala kuncishisiwe. Futhi lokhu kuboniswe eziphumeleni ezibheka ukuqhuma kwe-defibrillator namanani okufa okuzumayo. UDkt. Ommen: Iphethini yokudluliselwa ngezizukulwane ye-hypertrophic cardiomyopathy yi-autosomal dominant, okusho ukuthi ngamunye wezingane zomuntu onayo i-HCM unethuba elilinganayo le-50/50 lokudluliselwa lesi sifo. Siqondise ukuhlolwa kwabo bonke abantu abaseduze, okuyihlolwa le-genetic noma ukubhekwa kwe-echocardiographic. Uma umndeni ukhethe ukusebenzisa i-echocardiography njengesihluthulelo sabo sokuhlola, siqondise ukuthi abantu abadala abaseduze bahlolwe njalo eminyakeni emihlanu. Abantu abaseduze abasebasha noma abagijimi, ngokuvamile siwahlola njalo ezinyangeni eziyi-12 kuya kweyi-18. UDkt. Schaff: I-Septal myectomy iyalapha izimpawu ze-hypertrophic cardiomyopathy uma iqeda ukuvimba. Kepha, impela, abantu basene-hypertrophic cardiomyopathy, basadinga ukulandelelwa yidokotela wabo ngezinye izinkinga ezihlobene ne-hypertrophic cardiomyopathy. Kepha ngethemba, bayakhululeka ekuphumeni komoya, ubuhlungu besifuba, noma ukukhanya ekhanda okuholela ekuhlinzweni. Ulwazi Olwengeziwe Ukukhathalelwa kwe-hypertrophic cardiomyopathy e-Mayo Clinic Ukwelashwa kwe-ablation I-Implantable cardioverter-defibrillators (ICDs) I-Pacemaker Ividiyo: I-Septal myectomy ne-apical myectomy Bonisa olunye ulwazi oluhlobene Cela i-aphoyintimenti

Xhumana nabangane nomndeni wakho noma iqembu lokusekela. Ungase uthole ukuthi ukukhuluma nge-hypertrophic cardiomyopathy nabanye abantu abasesimweni esifanayo kungakusiza. Kubalulekile futhi ukulawula ukucindezeleka komzwelo. Ukuzivocavoca kakhulu nokwenza ingqondo kuyizindlela zokunciphisa ukucindezeleka. Uma unesizungu noma ukucindezeleka, khuluma neqembu lakho lokunakekelwa kwezempilo ngezindlela zokusiza.